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Cardiovascular disease in the mother and fetus incentives


Weight and body weight in the average age of the child's growth has reached Smilax best. Birth weight, fetal growth is a comprehensive index,mbt cipő, it sets a comprehensive head circumference,herve leger sale, length and fat reserves, and other factors more in-depth study of fetal growth and adult blood pressure between the different indicators of relationship, we analyzed 449 are men and women around the age of 5O-year-old resident . These residents are born in a hospital in Preston,mulberry bags uk, the newborn observed and recorded in great detail. Table 5 is the placental weight and birth weight in different populations, the average systolic blood pressure. Placental weight and birth weight with the opposite relationship between systolic blood pressure,diesel store paris, high birth weight population than the minimum systolic blood pressure by about 10mmHg,######## oakley sunglasses, and the highest crowd placental weight, systolic blood pressure than Table 5 - different birth weight and placental weight average population systolic blood pressure depression: Figures in brackets, for example number; sample of men and women aged 46 to 54. Were higher than the lowest group of about 12mmHg. By gestational age, current body mass index and the amount spilled drink after adjustment factors, these trends were not affected. The same clinical placental weight and adult hypertension. Table 5 also shows that, while under treatment for hypertension in the placental weight> 1.5 pounds is the proportion of groups in placental weight <1. O lb group in the proportion of 3,7 times. A recent study on 405 children of 4 years, there have been similar results. It is noteworthy that the vast majority of high systolic blood pressure of Preston's birth weight is not particularly low, while living in the normal range, but a large placenta. Placenta with a focus on an interesting feature of the child's head circumference is not commensurate with the length, it is too short. Now known animal embryo's response to hypoxia is a moderate cost of other organizations to ensure that cerebral blood flow and myocardial blood supply. It follows the existence of human fetal growth in a similar mechanism is not far-fetched. This is probably the placenta overweight children into a feature length, short storm. Causes the placenta is too large is not yet clear. The figures on Preston, middle and high social status of the mother's placenta overweight rate was only 7%, while the low social status were as high as 24%. Oxford recently born 8684 study of newborns with iron deficiency anemia is associated with placental weight. Shows that poor nutrition may be one of the causes of the placenta is too large. Cardiovascular disease research during the same time, the situation on the early growth and adult obstructive airway diseases in the study is in progress. Is now clear evidence that obstructive respiratory disease with fetal, infant lung development when rapid growth retardation. Infancy with acute respiratory tract infection. Conclusion The study shows that fetal, infant mortality, growth retardation and adult disease concentric to known cardiovascular risk factors closely related to the level of exposure. The question is how to explain this relationship, the extent to which it can be regarded as a causal link. Broadly speaking, there are three possible explanations. The first explanation is that birth weight and adult cardiovascular disease adversely affected by the same environmental factors. Ecological data separated the point of view, this interpretation may be established, but it is with the individual's prospective study findings. In Hertfordshire County, birth weight and current weight has nothing to do with social status. Indicators of early growth associated with adult risk factors exist in the social status of the population. Moreover, if fetal, infancy, poor environment, through a series of children and adolescence the cumulative effect of adverse effects caused by increased levels of risk factors in adults should be able to find risk factors and adult height between the negative relationship. However, the situation has been found with the early growth and adult blood pressure, the relationship between fibrinogen concentrations independent of height. The second explanation is that genetic factors. This effect manifested itself in the early growth retardation, and diseases of aging compared with adult. In other words, low birth weight determined genetically determined cardiovascular disease with the same or closely related genes. This explanation is not necessarily correct. Because birth weight does not seem primarily by genetic control, lots of people have not yet found from the relevant decisions of important genetic factors in cardiovascular disease, sufficient evidence. We believe that early growth retardation with adult disease risk associated with reason: a critical period of growth and development, the result of poor environment on the individual's physiology and metabolism of long-term effects. This may explain why the known adult risk factors for cardiovascular disease, the predictive ability is poor. Of course, this does not mean that the living conditions of adulthood is not important. Further research is focused on the environmental factors affecting fetal growth and infant characteristics and its specific metabolic programming when asked. At the same time, animal experiments are not the line is not rebellious, is actively through direct control of animal embryonic environment, and use of effective factors on the mother, ultrasound technology research all aspects of fetal development. (JEpidemlo] CommunityHealth1992; 46:81 - i1 (English) Wang Debin translation)
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